Pacientes produtores ativos de saúde (prosumo)

Essa avalanche de informações e conhecimento relacionada à saúde e despejada todos os dias sobre os indivíduos sem a menor cerimônia varia muito em termos de objetividade e credibilidade. Porém, é preciso admitir que ela consegue atrair cada vez mais a atenção pública para assuntos de saúde - e muda o relacionamento tradicional entre médicos e pacientes, encorajando os últimos a exercer uma atitude mais participativa na relação. Ironicamente, enquanto os pacientes conquistam mais acesso às informações sobre saúde, os médicos têm cada vez menos tempo para estudar as últimas descobertas científicas ou para ler publicações da área - on-line ou não -, e mesmo para se comunicar adequadamente com especialistas de áreas relevantes e/ou com os próprios pacientes. Além disso, enquanto os médicos precisam dominar conhecimentos sobre as diferentes condições de saúde de um grande número de pacientes cujos rostos eles mal conseguem lembrar, um paciente instruído, com acesso à internet, pode, na verdade, ter lido uma pesquisa mais recente do que o médico sobre sua doença específica. Os pacientes chegam ao consultório com paginas impressas contendo o material que pesquisaram na internet, fotocópias de artigos da Physician's Desk Reference, ou recorte de outras revistas e anuários médicos. Eles fazem perguntas e não ficam mais reverenciando a figura do médico, com seu imaculado avental branco. Aqui as mudanças no relacionamento com os fundamentos profundos do tempo e conhecimento alteraram completamente a realidade médica. Livro: Riqueza Revolucionária - O significado da riqueza no futuro

Aviso!

Aviso! A maioria das drogas psiquiátricas pode causar reações de abstinência, incluindo reações emocionais e físicas com risco de vida. Portanto, não é apenas perigoso iniciar drogas psiquiátricas, também pode ser perigoso pará-las. Retirada de drogas psiquiátricas deve ser feita cuidadosamente sob supervisão clínica experiente. [Se possível] Os métodos para retirar-se com segurança das drogas psiquiátricas são discutidos no livro do Dr. Breggin: A abstinência de drogas psiquiátricas: um guia para prescritores, terapeutas, pacientes e suas famílias. Observação: Esse site pode aumentar bastante as chances do seu psiquiatra biológico piorar o seu prognóstico, sua família recorrer a internação psiquiátrica e serem prescritas injeções de depósito (duração maior). É mais indicado descontinuar drogas psicoativas com apoio da família e psiquiatra biológico ou pelo menos consentir a ingestão de cápsulas para não aumentar o custo do tratamento desnecessariamente. Observação 2: Esse blogue pode alimentar esperanças de que os familiares ou psiquiatras biológicos podem mudar e começar a ouvir os pacientes e se relacionarem de igual para igual e racionalmente. A mudança de familiares e psiquiatras biológicos é uma tarefa ingrata e provavelmente impossível. https://breggin.com/the-reform-work-of-peter-gotzsche-md/

sábado, 9 de setembro de 2017

John Read: Electroshock

https://www.madinamerica.com/2017/09/john-read-science-evidence-electroshock/

John Read: What the Science and Evidence Tell Us About Electroshock

 

In this episode we discuss:

  • How Professor Read became interested in psychology, partly because of difficulties in his younger years and he wanted to understand those experiences
  • That his first experiences with patients in a psychiatric ward would be that people often wanted to share traumatic experiences, but that the psychiatrists didn’t seem that interested
  • That, by and large, mental health services around the world prefer to count symptoms and to medicate rather than to understand what has happened in a person’s life
  • How John came to have an interest in and research the efficacy and safety of Electroconvulsive Therapy (Electroshock)
  • That ECT is designed to induce a grand mal seizure and it started as a treatment for people diagnosed as schizophrenic
  • That the justification in the 1940s was that schizophrenics did not suffer with epilepsy and epileptics did not suffer with schizophrenia, so psychiatry made the leap to inducing epileptic seizures as a ‘cure’ for schizophrenia
  • That nowadays it is not used for people labelled as schizophrenic but it is most often used for treating depression
  • How actually it is not the diagnosis that is the best predictor of who gets ECT, it’s age and gender
  • Women aged over 60 are twice as often given ECT as men, and people over 60 are given it 2-3 times more often as those under 60
  • That the other rationale given for ECT treatment is the tendency for ECT to obscure traumatic memories because of memory loss
  • That the science and evidence tells us that after 70 years there has never been a single study showing that ECT is better than placebo beyond the end of the treatment period
  • That placebo in this sense is like sham surgery, the anaesthetic is given but not the electricity
  • That during the treatment (usually 3-4 weeks and an average of 8-10 sessions) roughly a third of those treated gain some lift of mood but that even for this minority of responders, the effect wears off after a few weeks
  • That this explains why some people will give anecdotal evidence that ECT saved their life and that they tend to have repeated treatments because they want the same life of mood
  • That the method used to assess success of the procedure is most often a rating scale or a ‘clinical judgement scale’ and these methods are open to bias
  • That there is not a single study that has ever shown that ECT can ‘prevent suicide’ when compared to placebo, the claims that it can are based on anecdotal evidence
  • That Earnest Hemingway killed himself shortly after receiving ECT saying “it was a brilliant cure, but unfortunately we lost the patient”
  • That there are temporary effects such as headaches after the procedure, but the enduring difficulties are often with memory loss which can be short term or longer term memories
  • Roughly a third of people will have serious, debilitating and ongoing memory loss which is caused by the brain damage caused by ECT
  • That the Guardian newspaper reported in April 2017 that ECT use was increasing in the UK but that their figures were wrong
  • That a third of psychiatrists will use ECT, a third will only use it after other options have been explored and a third will not use it under any circumstances
  • That ECT can get catatonic people moving and speaking but it is not difficult to artificially stimulate mood and it should not be seen as a cure
  • That there haven’t been any placebo controlled trials of ECT since 1985 and that was the last of only four that have ever been done that compared ECT with placebo after the end of treatment
  • How the fact that we do not have any successful trials showing that ECT is effective should mean that psychiatry either puts effort into proper research or that the procedure should be stopped
  • That John feels that eventually we will look back at ECT in the same way that we now view lobotomy, blood letting, rotating chairs and the like
  • How the principle should be informed consent and that people should be able to get treatment that they feel will help them but only if they know fully the risks and benefits and if they have been offered alternatives
  • There is a low but signifiant death rate from ECT, partly down to the general anaesthetic and partly due to cardiovascular failure because of the induced seizure but this death rate is never mentioned to potential patients
  • That it is probably down to the placebo effect of having attention and a procedure that expectations are created and hope is raised
  • That there is effort being put now into transcranial magnetic stimulation (TMS) and people can actually shock themselves using this method
  • That if we have large numbers of people walking round depressed, we really need to start asking questions about our society rather than trying to artificially eradicate those feelings
  • That John’s view is that depression is largely cause by depressing things happening to people rather than because of depressive illness and assuming that we can identify the parts of the brain that are ‘diseased’

 

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